Planning for Prostate Cancer Research
Expanding the Scientific Framework & Professional Judgement Estimates
Harold E. Varmus, M.D., Director National Institutes of Health Submitted June 1999

Section 4 of 13

Prostate Cancer Research Activities

The following are intended as a capsular summary of NIH's range of activities in prostate cancer research, elaborated on in this report:
The Cancer Genome Anatomy Project

The CGAP is an interdisciplinary program to establish information, reagents, and technological tools needed to decipher the molecular anatomy of a cancer cell. The overarching purpose of the CGAP is the comprehensive molecular characterization of normal precancerous and malignant cells. The Human Tumor Gene Index was initiated in May 1998 with the initial goal of identifying genes expressed during development of five major cancer sites - breast, colon, lung, ovary, and prostate. Over 80 cDNA libraries have been prepared and more than 300,000 partial cDNA sequences have been generated. To date, about 10,000 putative new genes have been discovered as a result of the CGAP. All CGAP data and reagents are made available to the research community without restriction via the CGAP website located at http://www. ncbi.nlm. nih.gov/ncicgap/.

To begin to examine gene expression profiles during human prostate cancer progression we prepared and sequenced clones from 20 cDNA libraries. We used a range of prostate-derived tissue sources, including microdissected cells (normal epithelium, prostatic intraepithelial neoplasia, invasive tumor, metastatic tumor), bulk tissue samples (normal, tumor), and matched normal and tumor prostate cell lines. A total of 40,637 sequence reads were performed which identified 7,677 unigene clusters expressed in normal or cancerous prostate tissues, 354 novel human genes, 146 prostate specific genes, 389 prostate unique genes, and 400 candidate differentially expressed genes. Comparison of cDNA library sequences derived from microdissected cells, cell lines, and bulk tumor libraries identified both an overlapping and distinct population of expressed genes from each group. Library normalization increased both the diversity of sequences and rate of novel gene discovery.


The Prostate Cancer Progress Review Group

The NCI has supported and continues to support a very wide range of basic, clinical, and population-based research projects to elucidate the causes and biology of prostate cancer and to develop strategies and technologies for preventing, detecting, diagnosing, and treating prostate cancer. New data suggest that the application of these research results may save lives, as evidenced by the declining mortality rate for prostate cancer among some populations. This ongoing research effort has provided a wealth of new scientific opportunities that if pursued may further advance our understanding of prostate cancer and our ability to care for men with this disease and those at risk of it. The NCI Director constituted the Prostate Cancer Progress Review Group (PRG) that began its work in April 1997. The Prostate PRG is one of two pilot groups that were established to help NCI sharpen the focus of its large, site-specific research programs. The other PRG focused on breast cancer. The PRGs are part of NCI's new overall planning framework, which also relies on Working Groups and Program Review Groups for evaluation, and planning of the research portfolio.

Specifically, the PRG was given six charges: 1) Identify and prioritize scientific needs and opportunities that are critical to hasten progress against prostate cancer. 2) Review an NCI-prepared portfolio analysis of the current research program. 3) Review recommendations from the research community generated through Prostate Cancer Roundtable meeting and Expert Panel reports. 4) Review recommendations from the prostate cancer advocacy community and from stakeholders. 5) Define and prioritize the research agenda. 6) Develop an action plan encompassing both operational and strategic components of the NCI's prostate cancer enterprise, using the current research program as the baseline for recommended actions.

Members of the PRG were selected from among prominent members of the scientific, medical, industrial, and advocacy communities to represent the full spectrum of expertise needed to develop comprehensive recommendations on NCI's prostate cancer research agenda. The membership was also selected for its ability to take a broad view in identifying and prioritizing scientific needs and opportunities that are critical to advancing the field of prostate cancer research. The full report of the Prostate Cancer Progress Review Group can be read on line at http://wwwosp.nci.nih.gov/Planning/PRG/default.htm A hard copy can be ordered by calling 1-800-422-6237, or by sending an e-mail message to nciosppcaprg-r@mail.nih.gov.


The Process of Reviewing the Prostate Cancer Portfolio

The PRG assessed the current NCI research portfolio in prostate cancer. The aim of the portfolio review was to describe the ongoing NCI prostate cancer research effort, which would then serve as a baseline for the PRG in formulating its recommendations. The NCI prostate cancer research portfolio was reviewed within the context of scientific questions and/or research areas. It became apparent during the Portfolio Analysis that if the NCI could develop a Common Scientific Outline that could be used for any specific cancer for comparing research between scientific organizations, this would be very beneficial to NCI and the cancer community at-large. The PRG, in addition to its main charge, played a critical role in providing a basis for the NCI to build this new and important tool, which will provide a standardized way to scientifically categorize NCI supported research. This scientific categorization will allow for thoughtful prioritization of research by identifying strengths, weaknesses, and trends within the NCI research portfolio. The Common Scientific Outline may be applied to each type of cancer individually (e.g., prostate, breast, lung, ovarian, etc.) to identify gaps in research, commonalities among cancer research fields that may be capitalized upon, or duplicative efforts that could be streamlined. A concrete example is the current collaboration between the NCI and the Department of Defense (DoD). The DoD is piloting the NCI Common Scientific Outline within the DoD's recent Prostate Cancer Research Program Announcement. The information from this pilot should provide the basis for future use of a Common Scientific Outline by both agencies. This could allow the NCI and DoD, for the first time ever, to compare effectively their individual research portfolios and to identify those areas that are undersupported or instances of duplication of effort between the two agencies.


Identification of Needs and Resources

The PRG used the portfolio analyses as a "baseline" in formulating their recommendations. Throughout their deliberations, the PRG relied extensively on input from outside experts. Key sources of information included:
Overall Results of the PRG Analysis of the NCI Portfolio in Prostate Cancer

After reviewing the portfolio of grants funded by NCI, the PRG estimated that there was over $82 million in 1997 supporting research in prostate cancer. While the process of attribution of many scientific projects was difficult - funded research projects often address diverse research topics as well as cancers in multiple tissues - the PRG was able to identify the major focus of projects and sorted them according to the major research areas. It is important to note that some projects overlap topic areas and are found in two or more categories. Epitomizing the issue of overlap are animal models for prostate cancer. Reliable and robust animal models for prostate cancer are recognized by all concerned as being of critical importance, and so the PRG dealt with this as a separate category. However, the models are being used to study biology, detection, diagnosis, prevention, and therapy, thus making it difficult to code a given project in only one category.

Projects related to prostate cancer conducted within the NCI Intramural Research Program as well as those sponsored by the Extramural Research Program were included in the portfolio review conducted by the Prostate Cancer PRG. In the sections that follow, an attempt is made to convey the breadth of the NCI portfolio in prostate cancer research.


Portfolio in Biology and Epidemiology

Understanding the molecular genetic changes associated with progression from pre-malignant to malignant states is essential to interrupt the process. The overall emphasis in research supported in this area is to understand how hormonal, extracellular matrix and possibly infectious factors modulate the expression of specific genes and, more importantly, the functional significance of these genetic changes. Investigations include: the delineation of genetic events in the development of prostate cancer; the role of sex hormones, various growth factors, their receptors and binding proteins in the malignant transformation; and the role of cell-cell and cell-matrix interactions in prostatic carcinogenesis

One of the major challenges in understanding the pathogenesis of prostate cancer continues to be the development of relevant models that mimic changes that occur in human prostatic neoplasms. Currently available models to study prostatic carcinogenesis include carcinogen-induced or transgenic animal models, normal prostatic epithelial cells exposed to various transforming agents in vitro, and use of human prostatic tissues (normal, benign and malignant).

A high proportion of indolent lesions characterizes prostatic neoplasms while only a small percentage of these lesions become invasive. Since it is not possible to predict the biological potential of these lesions based on histology alone, molecular mechanisms that distinguish non-invasive from aggressive lesions is a research area of high priority. Hormones and other signal transducers contribute to the complex cascade of the metastatic process including cell adhesion and motility, matrix degradation and angiogenesis.

Classical epidemiologic studies have identified heritability, i.e., familial clustering, positive family history, and Mendelian inheritance patterns, as one of the determinants of prostate cancer. In extending these findings while the search for predisposing gene(s) continues, currently supported investigations are pursuing several research directions. Future challenges will include elucidating the effects of environmental modulation of gene expression and gene-environment interactions and assessing risk of combined factors, such as a heritable component, intrinsic host and behavioral patterns, and environmental exposures.

Earlier studies have considered various environmental, biological, and lifestyle factors with conflicting results, often due to methodological issues. As molecular technology provides the capability for precision and increased power in epidemiologic studies, research will be needed to address relevance, validity, and applicability of molecular determinations in populations. Utilizable biological specimen banks will be invaluable. Most limiting and complex are the unanswered questions regarding the identification of influential environmental exposures.


Portfolio in Preventing Prostate Cancer

There is a 100-fold variation in prostate cancer incidence worldwide. The factors involved in this extreme variation of prostate cancer incidence must be investigated and identified to allow development of rational prostate cancer prevention strategies. Although research on hormonal factors of prostate cancer is the currently favored model, research on other modulable exposures, for example selenium intake, will require further investigation.

Chemoprevention of prostate cancer development is an important research area given the likelihood that modulable exposure (dietary factors, carcinogens etc.) may explain the 100-fold variation in prostate cancer incidence observed worldwide. There will continue to be a substantial need for developing cellular models for screening of agents and, in addition, reliable animal models for testing new agents before use in human subject clinical trials.


Portfolio in Detection and Diagnosis of Prostate Cancer

The effect of screening on prostate cancer mortality statistics is unknown. The primary method of screening for prostate cancer, at present, is by serum analysis for prostate-specific antigen (PSA). Elevation of PSA may indicate prostate cancer; however, several other common benign conditions, including Benign Prostatic Hyperplasia (BPH), are known to be associated with an elevated PSA. The widespread measurement of Prostate Specific Antigen (PSA) levels in blood has led to an increased detection rate for prostate cancer. Approximately 30 percent of early stage disease will progress to clinically relevant disease within the lifetime of the patient. Thus, there is a critical need to be able to identify those patients at risk of progression who would benefit from aggressive therapy while sparing low-risk patients the morbidity resulting from aggressive treatment of indolent disease.

The metastatic potential of prostate tumors is determined by the capacity of the tumor cells to escape the environment of the prostate gland, to survive distribution throughout the body, to implant at a distant site and to stimulate the new environment to produce the necessary vascularization and metabolic conditions to facilitate cellular proliferation. Identifying molecular alterations in tumor cells that reflect the tumor cells acquisition of any of these biological capacities would identify those patients at highest risk of metastatic disease.

Many prostate cancer patients initially respond to hormone therapy, only to fail with the appearance of recurrent disease that is no longer responsive to androgen or androgen analogs. The biological basis for androgen insensitivity is not well understood; significant efforts are being made to determine the role of the androgen receptor in the disease process. Extremely sensitive molecular technologies detect prostate specific mRNAs, such as PSA mRNA, in the blood and bone marrow in a subset of patients with organ confined disease. Initial studies have shown that patients with extraprostatic cells following surgery or radiation therapy are at high risk of treatment failure. Continuing studies are attempting to identify markers specifically associated with prostate cancer cells that will improve the specificity of the assay.

Prostate cancer is a heterogeneous disease with apparently independent foci of cancer scattered throughout the gland. The cancer foci have different malignant potentials and do not pose equal risks for the patient. Heterogeneity confounds the interpretation of prostate biopsies since it is not possible to be certain that the most clinically relevant foci of cancer have been detected.

Families have been identified in which the number of family members afflicted with prostate cancer is significantly higher than might be expected based on the rate of disease in the entire population. These families are particularly characterized by the early age of disease onset.

The incidence and severity of prostate cancer varies in different ethnic populations. African American men are more than twice as likely to die of prostate cancer than Caucasian men. In African American men, prostate cancer is also generally more advanced at the time of diagnosis. The biological basis for this racial difference in prostate cancer is not understood.

There are a number of high-priority research and development areas for imaging modalities in the detection and diagnosis of prostate cancer. Ultrasound technology continues to be an important tool in the detection of prostate cancer. Another focus of prostate imaging research is the use of magnetic resonance imaging and spectroscopy that have advanced technologically to the point where large-scale clinical trials could be initiated for the prostate.


Portfolio in Treating Prostate Cancer

Clinical management of prostate cancer has, until recently, been restricted by a limited armamentarium of classical modalities - surgery, radiation, androgen deprivation, and (minimally active) cytotoxic chemotherapy. There remains a need for a broad program for testing novel approaches, compounds, and targets, for prioritizing these, and for answering the practical questions necessary for preparing these for clinical testing.

Management of prostate cancer presents some unique complexities for oncologists and other clinicians. One set of difficulties is the inability to reliably distinguish in advance those tumors whose behavior will be aggressive from the larger subset of indolent tumors, the very long intervals before clinical trials become mature, and the fact that good surrogate endpoints are not yet known. The entire clinical trials capacity for prostate cancer needs to be expanded since the participation of large numbers of patients can reduce somewhat the time to definitive analysis of these clinical trials. Moreover, only about 30 percent of patients diagnosed with limited stage prostate cancer will eventually progress to have clinically relevant disease. This complicates profoundly the evaluation of therapies because the majority of patients whose clinical course is inherently benign can overwhelm recognition of treatment effects in the minority.

Progress in clinical therapy of other malignancies has generally accompanied our capacity to refine both risk classification and prediction of therapeutic response or non-response. This has made it possible to test treatments in more homogeneous clinical cohorts, with the result that ineffective therapy can be abandoned in favor of earlier testing of novel approaches. The exploration of defects in cellular regulatory, signaling, and apoptosis pathways in prostate cancer as determinants of hormonal resistance is likely to provide a productive field for testing in the clinical arena in the near future.

Clinical intervention trials, both therapeutic and chemopreventive, have been hampered by the insensitivity of clinical evaluations for prostate cancer, the unwillingness of patients and clinicians to undertake repeated uncomfortable invasive procedures, and the lack of reliable non-invasive methodologies for response assessment. This is an area in which the research portfolio is currently virtually empty. Imaging technologies per se, however, make up an extremely active field of research and development, with the potential to dramatically improve our evaluation of potential chemopreventative approaches and of local therapies such as various modalities of radiation, cryosurgery, etc. and of the role of adjunctive hormonal, anti-invasive, or other therapy.


Portfolio in Prostate Cancer Survivorship and Morbidity

The Prostate Cancer Outcomes Study (PCOS) is a major NCI study being conducted by six out of the 10 registry organizations that participate in the NCI's Surveillance, Epidemiology and End Results (SEER) Program. The objective of PCOS is to evaluate recent diagnostic and treatment practice patterns among men diagnosed with prostate cancer in order to evaluate the prevalence of long-term urinary, bowel, and sexual function complications subsequent to initial treatment. It is recognized that more needs to be done in managing incontinence and sexual dysfunction.

Prostate cancer, as the most commonly diagnosed cancer among men in the U.S., exerts an enormous impact on patients. In addition to the expense of an estimated $5 billion spent annually in direct medical care costs there are questions about quality of life. The NCI portfolio includes support for the development of instruments to assess quality of life and interventions to increase health-related quality of life of prostate cancer patients. Symptom management specific to the sequelae of prostate cancer therapy should be examined more broadly.


Portfolio in Prostate Cancer Surveillance

Diet is monitored among the general population, using nationally representative samples, and among special populations defined by gender, age, race, and ethnicity. Selected research priorities include monitoring trends in intakes of specific food groups and nutrients; identifying food sources of nutrients; and assessing demographic and other factors that affect changes in dietary behaviors over time.

NCI monitors the dissemination of state-of-the-art diagnostic and treatment modalities. This research is being conducted using survey data, tumor registry information from the Surveillance, Epidemiology, and End Results (SEER) Program, and from administrative data sources such as Medicare claims linked with SEER data. Innovative approaches are being developed to relate current and future trends in PSA usage to prostate cancer incidence and mortality. Other innovative methodologic approaches that are being used to model trends in prostate cancer include incidence-based mortality and change point techniques.


Portfolio in Scientific and Public Resources

The National Cancer Act of 1971 explicitly instructed the Director of the NCI regarding communication, and the NCI sees communication as a critical component of the NCI's mission. The NCI not only engages in education and communication but also supports research into how to educate and communicate effectively. Many of these efforts are crosscutting, but some relate to specific cancer sites.

Cancer center support grants (CCSG) provide support to the peer-reviewed research base of a cancer center within the parent institution. In addition, development grants (R21) for new research programs in prostate cancer are exploratory-developmental grants to promote the development of institution-wide multidisciplinary research programs in prostate cancer.

Specialized Programs of Research Excellence (SPOREs) focus on translational research, i.e., the bi-directional exchange between basic and clinical science in order to move basic research findings from the laboratory to applied settings involving patients and populations. The ultimate goal of the SPORE program is to bring novel ideas that have the potential to reduce cancer incidence and mortality, improve survival, and to improve the quality of life to clinical care settings. At present, there are three Prostate Cancer SPOREs: at the Baylor College of Medicine, at the Johns Hopkins Cancer Center, and at the University of Michigan Cancer Center. Among the prostate cancer research resources supported through the NCI are the Community Clinical Oncology Program (CCOP). These are organizations comprising one or more facilities (e.g., community hospitals, clinics, doctors' offices) which together function as an infrastructure for the implementation of clinical trials in the community.

Human tissue resources include the Cooperative Human Tissue Network, a tissue procurement network that provides tissues on request to investigators. Approximately 10 percent of the tissues provided through this network are prostate. This effort is spread over the four sites: Case Western University, Ohio State University, the University of Alabama, and the University of Pennsylvania. The Developmental Therapeutics Program (DTP) uses contract resources to discover, test and develop anti-cancer agents. DTP's developmental resources provide for chemical synthesis, formulation, pharmacology, and toxicology testing for promising agents through the NCI's Decision Network Committee.


Recommendations of the PRG

While the dollar amount and number of grants attests to a substantial commitment of NCI to research in prostate cancer, the PRG nonetheless identified gaps in the research agenda and major new opportunities that could move the pace of research in prostate cancer forward over the coming decade. The PRG examined the research portfolio and developed a ranked listing of priorities for research and the resources needed to support an expanded program of research. Their recommendations can be summarized as follows:
New Initiatives in Prostate Cancer Research

Partially in response to the report of the Prostate Cancer PRG and also as a part of its normal planning process, the NCI has begun a number of new initiatives with relevance to research in prostate cancer. NCI is also seeking to communicate more effectively both new and long-standing initiatives to the extramural research community to stimulate their increased involvement in the battle against prostate cancer. Toward that end, NCI has recently added to its website a page describing initiatives applicable to prostate cancer research these initiatives. The page can be accessed at http://www.nci.nih.gov/prostate.html. It contains descriptions of initiatives, hotlinks to other relevant sites, and NCI contact information. The topics covered in this webpage are:
Clinical Trials in Prostate Cancer

In 1997 and 1998, NCI funded (in total or in part) 246 clinical trials in prostate cancer including 80 Phase III studies (clinical trials involving new interventions closest to approval) and 37 Phase I trials (representing the newest potential therapeutic agents for prostate cancer). At the NIH Clinical Center 13 NCI trials in prostate cancer are underway. NCI clinical studies in prostate cancer have significant African American participation. In one NCI study 14.7 percent of men enrolled onto NCI sponsored prostate cancer treatment trials are African American while 10.3 percent of Americans diagnosed with prostate cancer are African American.

NCI's ongoing Prostate Cancer Prevention Trial (PCPT) involves 18,000 healthy men over the age of 55 to determine if the drug finasteride can prevent prostate cancer. NCI's ongoing Prostate, Lung, Colorectal, and Ovarian Cancer Screening Trial (PLCO) is assessing the efficacy of prostate cancer screening. NCI is sponsoring two trials in which "watchful waiting" is being compared in terms of outcome with surgical removal of the prostate and with radiation therapy. NCI has conducted a large interview-based study of prostate cancer in African Americans and Whites. NCI funded or co-funded 11 prostate cancer epidemiologic studies beginning in September 1995 with study durations of 3-5 years. Eight of the 11 studies include an assessment of risk factors among African American men.

Prostate cancer incidence is higher among African Americans (1987-1991; 163 per 100,000) than Whites (121 per 100,000). The disease generally occurs more frequently among Whites in the United States than in Europe. Asians, in comparison, have very low rates of occurrence (for China, less than one percent of the rate among African Americans). In the United States, mortality due to prostate cancer is two-fold greater among African Americans (52 per 100,000) than Whites (24 per 100,000) (Cancer Rates and Risks, 1996). NCI investigations are ongoing to identify risk factors for prostate cancer in several populations, including studies of African Americans and Whites in the United States and of Chinese in Shanghai.

The Human Genome Project has undertaken an effort to identify genes associated with the familial transmission of risk. The NCI plans studies to evaluate genetic determinants of susceptibility in the general population, including the impact of metabolic (e.g., polymorphism for 5-alpha reductase) and functional polymorphism (e.g., polymorphism in the androgen receptor). These studies will take advantage of the natural variation in polymorphism frequency between African American, White, and Asian populations. Vasectomy has been linked to prostate cancer among U.S. Whites, but several other studies provided inconsistent results. This remains an area of continued investigation. An etiologic link with benign prostatic conditions is also being investigated. Dietary fat has been linked to prostate cancer in several studies. This and other dietary factors (e.g., level of consumption of fruits and vegetables) are now being examined as risk factors in the African American, White, and Chinese study groups, providing an opportunity to assess risk over a broad range of dietary patterns.

The Cancer Therapy Evaluation Program (CTEP) is the program within the Division of Cancer Treatment and Diagnosis (DCTD) of the NCI. This program plans, assesses, and coordinates all aspects of clinical trials including extramural clinical research programs, internal resources, treatment methods and effectiveness, and compilation and exchange of data as it pertains to the development and evaluation of anticancer agents. Many of the compounds in early development or preclinical development will be suitable for studies in prostate cancer, since broad phase II solicitations typically include the common solid tumors, including prostate cancer.

Some agents specifically being developed for patients with prostate cancer include: Other agents that target angiogenesis and the metastatic phenotype including vitaxin, endostatin, Sugen 5416 which inhibits VEGF signaling through Flk-1, and thalidomide as well as newer generation matrix metalloproteinase inhibitors will be used in trials in prostate cancer and a number of new clinical trials will begin in 1999. Patients with hormone sensitive disease will be eligible for a planned trial of leuprokide to maximal PSA reduction and then maintenance thalidomide, with reinstitution of leuprolide when PSA rises, plus thalidomide if not previously received.

Pharmaceutical collaborators will be sponsoring prostate cancer studies with a number of agents that are being co-developed with NCI, including: antisense BCL-2 and Onyx 015, E1B deleted adenovirus.

Among the more interesting of the new approaches being studied in CTEP-sponsored trials in 1997 and 1998 are: vaccine trials involving fowlpox or vaccinia viruses that express PSA, thalidomide (as an anti-angiogenic agent) + combined androgen blockade, and a number of agents that may target critical signaling pathways, bryostatin-1, phenylbutyrate + all-trans-retinoic acid, flavopiridol, perillyl alcohol, and dolastatin -10.

CTEP anticipates initiating Phase I or II clinical trials with approximately 25 new agents or combinations in the next 12 to 18 months. In addition, 15 clinical trials in prostate cancer are in review and should be active shortly, including six Phase I, five Phase II, three-Phase III and one correlative science study. Overall, 40 to 50 clinical trials are likely.


Clinical Trials Restructuring and Genitourinary Cancers

The NCI is engaged in a major restructuring of its entire extramural large scale clinical trials program, its mechanism for carrying out the research with the greatest immediate effect on patient care. This restructuring, the most far reaching such reorganization in 15 to 20 years, is one outcome of extensive analysis and review of NCI programs by extramural scientists, patients, and advocates carried out over the last several years at the request of the NCI Director. NCI proposes to jump-start an expansion of clinically related research on urologic cancers by making these malignancies the focus of the set of pilot projects that together constitute a new framework for promoting and developing a rationale and innovative clinical research program for any and all types of cancer. A permanent ongoing series of nationally constituted State of the Science meetings in genitourinary cancers intended to identify needed resources (such as standardized outcomes endpoints), gaps in clinical research portfolios, and new clinical research opportunities. The recommendations and work products of these State of the Science meetings will be disseminated broadly to active investigators, specialty societies, advocacy organizations, and to the public via NCI information resources. It is anticipated that broad availability of this information will serve to stimulate innovative proposals for clinical trials in genitourinary malignancies, especially where important opportunities are identified.

1999 will see the establishment of a new entity that will accept developed clinical phase III trial proposals from any source and select all those that are considered of high merit to utilize the funding and resources of the NCI's clinical program and network. This Genitourinary Cancer Concept Review Committee will be nationally representative and composed of oncologists and Genitourinary physicians, clinical and laboratory scientists with special expertise related to genitourinary cancer, patients and patient advocates, biostatisticians, and some NCI staff. This committee will review, critique, and rank all proposals for randomized trials. The highest-ranked trials will be opened in the national network of qualified clinical investigators and physicians, and their patients, and will receive NCI funding for the scientific leadership and for reimbursement for research personnel costs on a per-case basis.

Another key aspect of the new framework (and a distinct departure from prior practice) will be the creation of a system that will provide all qualified investigators and eligible patients, regardless of their institutional or other affiliation, access to enrollment on any NCI supported phase III clinical trial in genitourinary cancers. Another new entity, a Cancer Trials Support Unit (CTSU) will be created to facilitate the transition from the old system to the new system and to provide a way to verify the accuracy of data arriving from doctors not affiliated with the study leadership, and for reimbursing the costs associated with recruitment of patients.

We anticipate that, taken as a package, this would be a much more comprehensive approach than the more conventional approach of increasing the number of centers of excellence, or adding consortia or Cooperative Groups. By opening up the clinical trials system to investigators and patients, these new initiatives should serve to expand substantially the pool of new approaches and clinical trials in prostate and genitourinary cancer, as well as providing the means to promote and make accessible to all patients the studies of the highest priority questions.


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